The vagus nerve does it all—tells your lungs to breathe, controls your heart rate, and even forms the foundation for a whole ...

Norepinephrine is also released from sympathetic nervous ... T-helper lymphocyte type 2 cells; TNF, tumour necrosis factor.

The regulation of vasoconstriction is also influenced by circulating catecholamines, such as epinephrine, norepinephrine ... as the method of cellular elimination in wounds The process of necrosis, ...

Her study found the glymphatic clearance was mediated by a hormone called norepinephrine and happened almost exclusively during the NREM sleep phase. But it only worked when sleep was natural.

Case 1. A. H., a 52-year-old Negro, was admitted to the hospital for treatment of malignant hypertension. On November 21, 1952, a left thoracolumbar sympathectomy was performed by a transthoracic ...

Fluid, electrolyte and mineral perturbations are prevalent features of tropical disease. Hemodynamic alterations, fever, nitrogen wasting, and changes in membrane transport and acid–base balance ...

The peripheral intravenous (PIV) catheter is the most used vascular access device for the administration of medications in hospitalized neonates, however 95% of PIV catheters are removed due to ...

Purpose. The efficacy and safety of granulocyte colony-stimulating factor (G-CSF) in critically ill patients with severe sepsis or septic shock were evaluated. Summary. The medical literature was ...

Eating certain foods, like those high in protein, and healthy practices like exercising and sleeping the recommended amount can help increase your body’s dopamine levels without medication ...

Terlipressin is considered the first-line agent in combination with 20–25% albumin. However, meta-analyses have shown comparative effects between norepinephrine and terlipressin for reversal of ...

Immune cells rely on cell-cell communication to specify and fine-tune their responses. They express an extensive network of cell communication modes, including a vast repertoire of cell surface and ...

Amyloid deposits can also directly cause myocyte necrosis by oxidative stress, and this can contribute further to systolic dysfunction. Moreover, deposits in the conduction tissue can affect ...